Basal Instinct: Persistence of Barrier Dysfunction

Caroline Chauché; Henry J. McSorley

doi:10.1016/j.immuni.2018.09.022

Basal Instinct: Persistence of Barrier Dysfunction

Caroline Chauché, Henry J. McSorley (Lead / Corresponding author)

Cell Signalling and Immunology

Research output: Contribution to journal › Short survey › peer-review

3 Citations (Scopus)

Abstract

In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype. In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype.

Original language	English
Pages (from-to)	590-592
Number of pages	3
Journal	Immunity
Volume	49
Issue number	4
Early online date	16 Oct 2018
DOIs	https://doi.org/10.1016/j.immuni.2018.09.022
Publication status	Published - 16 Oct 2018

ASJC Scopus subject areas

Immunology and Allergy
Immunology
Infectious Diseases

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

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10.1016/j.immuni.2018.09.022Licence: Elsevier User Licence

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title = "Basal Instinct: Persistence of Barrier Dysfunction",

abstract = "In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype. In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype.",

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AU - McSorley, Henry J.

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AB - In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype. In a recent issue of Nature, Ordovas-Montanes et al. (2018) used cutting-edge genomic, epigenetic, and interventional techniques to characterize the cellular ecosystem in allergic chronic rhinosinusitis. They showed that basal epithelial cells “remember” type 2 inflammatory stimuli to maintain a chronic allergic disease phenotype.

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Basal Instinct: Persistence of Barrier Dysfunction

Abstract

ASJC Scopus subject areas

UN SDGs

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