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Cyclosporin-induced renal vasoconstriction is augmented by frusemide and by angiotensin II in humans

    Research output: Contribution to journalArticlepeer-review

    Abstract

    To investigate the role of the renin-angiotensin-aldosterone system as a homoeostatic mechanism by examining whether mild activation of the renin-angiotensin-aldosterone system could enhance cyclosporin-induced renal vasoconstriction and hypertension.
    Original languageEnglish
    Pages (from-to)987-991
    Number of pages5
    JournalJournal of Hypertension
    Volume13
    Issue number9
    Publication statusPublished - 1995

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