Mechanism of mitochondrial dysfunction in diabetic sensory neuropathy

Keyphrases

• Insulin 100%
• Mitochondrial Dysfunction 100%
• Sensory Neurons 100%
• Diabetic Sensory Neuropathy 100%
• Mitochondrial Membrane Potential 80%
• Neurotrophin-3 (NT-3) 80%
• Diabetes 40%
• Gene Expression 40%
• Diabetic Neuropathy 40%
• Neurotrophic Support 40%
• Type I Diabetes 20%
• Structural Change 20%
• Significant Loss 20%
• Phosphoinositide 3-kinase (PI3K) 20%
• Downstream Targets 20%
• Transcription Factor 20%
• CAMP Response Element-binding Protein 20%
• Metabolic Pathway 20%
• Etiology 20%
• Most Common Form 20%
• ATP Synthesis 20%
• In Vivo Applications 20%
• Real-time Fluorescence 20%
• Peripheral Nervous System 20%
• Demyelination 20%
• Phosphoinositide 3-kinase Pathway 20%
• Video Microscopy 20%
• Degenerative Disease 20%
• Peripheral Nerve 20%
• Dieback 20%
• Etiological Factors 20%
• Axonal Degeneration 20%
• Distal Axons 20%
• Diabetic Rats 20%
• Mitochondria-associated Membranes 20%
• Mitochondrial Electron Transport Chain 20%
• Paranodal 20%
• Reducing Equivalents 20%
• Electron Transport Chain 20%
• Neuronal Mitochondria 20%
• Sensory Polyneuropathy 20%
• Membrane Polarization 20%
• Myelinated Fibers 20%

Neuroscience

• Sensory Neuron 100%
• Neuropathy 100%
• Mitochondrial Membrane Potential 80%
• Neurotrophin-3 80%
• Gene Expression 40%
• Phosphoinositide 3-Kinase 40%
• Electron Transport Chain 40%
• Diabetic Neuropathy 40%
• In Vivo 20%
• Cyclic Adenosine Monophosphate 20%
• Mitochondrion 20%
• Adenosine Triphosphate 20%
• Protein Binding 20%
• Transcription Factors 20%
• Peripheral Nervous System 20%
• Mitochondrial Membrane 20%
• Response Element 20%
• Nerve Fiber Degeneration 20%
• Axonal Degeneration 20%
• Peripheral Nerve 20%
• Polyneuropathy 20%
• Axon 20%