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Molecular pathways: is AMPK a friend or a foe in cancer?
D. Grahame Hardie
Cell Signalling and Immunology
Research output
:
Contribution to journal
›
Article
›
peer-review
148
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Citations (Scopus)
230
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Projects
(2)
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Dive into the research topics of 'Molecular pathways: is AMPK a friend or a foe in cancer?'. Together they form a unique fingerprint.
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Keyphrases
In Cancer
100%
Protein Kinase
100%
AMP-activated Protein Kinase
100%
Molecular Pathways
100%
Cell Proliferation
25%
Tumor Cells
25%
Cell Growth
25%
Cancer Development
12%
Proliferating Cells
12%
Quiescent Cells
12%
Eukaryotic Cells
12%
Selection Pressure
12%
Frequent Use
12%
Kinase Activation
12%
Metformin
12%
Glycolysis
12%
Glucose Uptake
12%
Human Cancer
12%
Cellular Energy Status
12%
Energy Homeostasis
12%
Pharmacologic
12%
Oxidative Metabolism
12%
Kinase Function
12%
Switch-off
12%
Anabolic Pathways
12%
Catabolic Pathway
12%
Salicylate
12%
Energetic Stress
12%
Glucose Sparing
12%
Medicine and Dentistry
Malignant Neoplasm
100%
Hydroxymethylglutaryl Coenzyme A Reductase Kinase
100%
Cyclic AMP Dependent Protein Kinase
100%
Adenosine Triphosphate
50%
Cell Proliferation
37%
Tumor Cell
25%
Cell Growth
25%
Cancer
12%
Adenosine Phosphate
12%
Glycon
12%
Cell Energy
12%
Glucose Uptake
12%
Homeostasis
12%
Salicylic Acid
12%
Salicylate Sodium
12%
Adenosine Diphosphate
12%
Energy Transfer
12%
Aerobic Metabolism
12%
Eukaryotic Cell
12%
Pharmacology, Toxicology and Pharmaceutical Science
Malignant Neoplasm
100%
Cyclic AMP Dependent Protein Kinase
100%
Hydroxymethylglutaryl Coenzyme A Reductase Kinase
100%
Adenosine Triphosphate
50%
Neoplasm
25%
Adenosine Phosphate
12%
Metformin
12%
Salicylate Sodium
12%
Adenosine Diphosphate
12%
Salicylic Acid
12%