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Morphine activation of mu opioid receptors causes disinhibition of neurons in the ventral tegmental area mediated by β-arrestin2 and c-Src
Fiona A. Bull
,
Daniel T. Baptista-Hon
,
Jeremy J. Lambert
, Wendy Walwyn
,
Tim G. Hales
(Lead / Corresponding author)
Research output
:
Contribution to journal
›
Article
›
peer-review
23
Citations (Scopus)
554
Downloads (Pure)
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Dive into the research topics of 'Morphine activation of mu opioid receptors causes disinhibition of neurons in the ventral tegmental area mediated by β-arrestin2 and c-Src'. Together they form a unique fingerprint.
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Keyphrases
μ-opioid Receptor
100%
Morphine
100%
C-Src
100%
Disinhibition
100%
Ventral Tegmental Area
100%
Arrestin2
100%
Inhibitory Postsynaptic Current
44%
δ Opioid Receptor
44%
Current Frequency
33%
Tyrosine Kinase
11%
Receptor-mediated
11%
GABAergic
11%
Negligible Effects
11%
Opioids
11%
Sensory Neurons
11%
Src Inhibitor
11%
MEK Inhibitor (MEKi)
11%
Spontaneous Inhibitory Postsynaptic Currents
11%
Reward Pathway
11%
DPDPE
11%
Receptor Neurons
11%
SL327
11%
Neuroscience
Morphine
100%
μ-Opioid Receptor
100%
Oncogene Src
100%
Disinhibition
100%
Ventral Tegmental Area
100%
Delta Opiate Receptor
44%
Tyrosine Kinase
11%
Receptor Tyrosine Kinase
11%
Sensory Neuron
11%
enkephalin[2,5 Dextro Penicillamine]
11%
MEK Inhibitor
11%