Skip to main navigation
Skip to search
Skip to main content
Discovery - the University of Dundee Research Portal Home
Home
Profiles
Research units
Research Outputs
Projects
Datasets
Theses
Activity
Press/Media
Research Facilities
Prizes
Search by expertise, name or affiliation
Multisite phosphorylation and the integration of stress signals at p53
David W. Meek
Research output
:
Contribution to journal
›
Review article
›
peer-review
180
Citations (Scopus)
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Multisite phosphorylation and the integration of stress signals at p53'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Phosphorylation
100%
DNA Damage
100%
Multisite Phosphorylation
100%
P53 Activity
100%
Stress Signaling
100%
Tumor Development
50%
Signaling Pathway
50%
Oncogene
50%
Mitosis
50%
Cell Death
50%
Apoptosis
50%
Hypoxia
50%
Response Change
50%
Amino Acids
50%
Phosphorylation Sites
50%
Phosphorylation State
50%
Tumor Protein p53 (TP53)
50%
Cellular Stress
50%
Sigma Phase
50%
Activation Domain
50%
Transcription Factor
50%
C-terminus
50%
DNA Binding
50%
Growth Arrest
50%
Transcription-independent
50%
Functional Domain
50%
Cellular Environment
50%
Viral Infection
50%
Response to Stress
50%
Cellular Growth
50%
Growth Suppression
50%
Metabolic Changes
50%
Covalent Modification
50%
Binding Function
50%
Latent Form
50%
Biochemistry, Genetics and Molecular Biology
P53
100%
DNA Damage
20%
Amino Acids
10%
C-Terminus
10%
Growth Inhibition
10%
Cell Death
10%
Transcription Factors
10%
N-Terminus
10%
Transcription
10%
Cytokine
10%
Cell Growth
10%
Transactivation
10%
Cell Stress
10%
S Phase
10%
DNA Binding
10%
Oncogene
10%
Programmed Cell Death
10%
Stress Signaling
10%
Immunology and Microbiology
P53
100%
DNA Damage
20%
Cytokine
10%
Carboxy Terminal Sequence
10%
Cell Death
10%
Cell Growth
10%
Transactivation
10%
Cell Stress
10%
Growth Inhibition
10%
Amino Terminal Sequence
10%
Transcription Factors
10%
DNA Binding
10%
Viral Disease
10%
Oncogene
10%
Programmed Cell Death
10%
Amino Acid
10%
Neuroscience
P53
100%
DNA Damage
20%
Transactivation
10%
Cell Growth
10%
Amino Acid
10%
Cell Death
10%
C-Terminus
10%
Oncogene
10%
DNA Binding
10%
Amino Terminal Sequence
10%
Transcription Factors
10%
Cell Stress
10%
Growth Disorder
10%
Programmed Cell Death
10%