Abstract
The contractility of the human uterus is under the fine control of a variety of interacting bioactive agents. During labour, the excitability of the uterus is drastically transformed in comparison with the non-labour state and is manifest at the membrane level via the activity of uterine ion channels. This article reviews the contribution of potassium (K+) channels to human uterine excitability. Experimental Physiology (2001) 86.2, 255-264.
Original language | English |
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Pages (from-to) | 255-64 |
Number of pages | 10 |
Journal | Experimental Physiology |
Volume | 86 |
Issue number | 2 |
DOIs | |
Publication status | Published - 2001 |