Signaling downstream of p38 in psoriasis

J. Simon C. Arthur; Joanne Darragh

doi:10.1038/sj.jid.5700280

Signaling downstream of p38 in psoriasis

J. Simon C. Arthur (Lead / Corresponding author), Joanne Darragh

Research output: Contribution to journal › Comment/debate › peer-review

22 Citations (Scopus)

Abstract

Psoriasis is an inflammatory skin disease characterized by infiltration of the skin by T cells and increased production of pro-inflammatory cytokines. Two recent reports show that the p38-activated kinases mitogen-activated protein kinase-activated protein kinase 2 and mitogen- and stress-activated protein kinase are activated in psoriatic skin and may contribute to the production of pro-inflammatory cytokines.

Original language	English
Pages (from-to)	1689-1691
Number of pages	3
Journal	Journal of Investigative Dermatology
Volume	126
Issue number	8
DOIs	https://doi.org/10.1038/sj.jid.5700280
Publication status	Published - Aug 2006

ASJC Scopus subject areas

Biochemistry
Molecular Biology
Dermatology
Cell Biology

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title = "Signaling downstream of p38 in psoriasis",

abstract = "Psoriasis is an inflammatory skin disease characterized by infiltration of the skin by T cells and increased production of pro-inflammatory cytokines. Two recent reports show that the p38-activated kinases mitogen-activated protein kinase-activated protein kinase 2 and mitogen- and stress-activated protein kinase are activated in psoriatic skin and may contribute to the production of pro-inflammatory cytokines.",

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AB - Psoriasis is an inflammatory skin disease characterized by infiltration of the skin by T cells and increased production of pro-inflammatory cytokines. Two recent reports show that the p38-activated kinases mitogen-activated protein kinase-activated protein kinase 2 and mitogen- and stress-activated protein kinase are activated in psoriatic skin and may contribute to the production of pro-inflammatory cytokines.

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JO - Journal of Investigative Dermatology

JF - Journal of Investigative Dermatology

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ER -

Signaling downstream of p38 in psoriasis

Abstract

ASJC Scopus subject areas

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